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  • Role of Pyroptosis in Inflammatory Bowel Disease (IBD): From Gasdermins . . .
    Pyroptosis is a pro-inflammatory cell death executed by gasdermin family proteins that involve the formation of pores on cells, recognition of danger signals, and release of pro-inflammatory cytokines IL-1 β and IL-18 Pyroptosis modulates mucosal innate immunity and enteropathogenic bacterial infection
  • Pyroptosis: Induction and inhibition strategies for immunotherapy of . . .
    The pore structures (with inner diameters ranging from 15 to 21 5 nm 17) in the cell membrane trigger pyroptosis, inducing osmotic cell swelling, membrane rupture, and the release of intracellular contents and cytokines (IL-1β and IL-18) to ultimately cause proinflammatory cell death 22
  • Mechanisms of Disease: Inflammatory Bowel Diseases
    In this article, we will review the mechanisms proposed to cause IBD from the genetic, environmental, intestinal barrier, and immunologic perspectives
  • IL-33 and IL-18 in Inflammatory Bowel Disease Etiology and . . . - Frontiers
    In this review we will examine two IL-1 family cytokines, IL-33 and IL-18, in the context of mucosal immunity and with a particular focus on the pathogenesis of IBD, a chronic inflammation of the intestinal mucosa
  • Role of interleukin-6-mediated inflammation in the pathogenesis of . . .
    Moreover, inflammatory mediators such as interleukin-6 (IL-6) are involved in the immunopathogenesis of IBDs IL-6 contributes to T helper 17 (Th17) differentiation, mediating further destructive inflammatory responses in CD and UC
  • Biological and clinical roles of IL-18 in inflammatory diseases
    Prevention of pyroptosis due to GSDMD pores can be provided by the ESCRT pathway b, Once released, it is typically bound and neutralized by IL-18 binding protein (IL-18BP), which is itself made
  • Frontiers | Role of Pyroptosis in Inflammatory Bowel Disease (IBD . . .
    Pyroptosis is a pro-inflammatory cell death executed by gasdermin family proteins that involve the formation of pores on cells, recognition of danger signals, and release of pro-inflammatory cytokines IL-1 β and IL-18 Pyroptosis modulates mucosal innate immunity and enteropathogenic bacterial infection
  • Inflammasome‐associated cell death: Pyroptosis, apoptosis, and . . .
    Inflammasomes are innate immune mechanisms that promote inflammation by activating the protease caspase-1 Active caspase-1 induces pyroptosis, a necrotic form of regulated cell death, which facilitates the release of intracellular proinflammatory molecules, including IL-1 family cytokines
  • Immunology of Inflammatory Bowel Disease: Molecular Mechanisms and . . .
    In this review, we explained the interactions among symbiotic flora, intestinal epithelial cells, and the immune system, clarified the operating mechanism of the intestinal immune system, and highlighted the immunological pathogenesis of IBD, with a focus on the development of immunotherapy for IBD
  • Molecular mechanisms and functions of pyroptosis, inflammatory caspases . . .
    Physical rupture of the cell causes release of the pro-inflammatory cytokines IL-1β and IL-18, alarmins and endogenous danger-associated molecular patterns, signifying the inflammatory potential of pyroptosis Here, we describe the central role of inflammatory caspases and pyroptosis in mediating immunity to infection and clearance of pathogens
  • What is Pyroptosis and the Underlying Molecular Mechanisms | Bio-Techne
    Pyroptosis is an inflammatory form of programmed cell death characterized by the release of pro-inflammatory cytokines IL-1β and IL-18 1,10 It is a process distinct from apoptosis and necrosis (Table 1) and has been observed in the setting of multiple diseases including, but not limited to, microbial infection 10, atherosclerosis and other
  • Pyroptosis and the Underlying Molecular Mechanisms - Novus Biologicals
    Pyroptosis is an inflammatory form of programmed cell death characterized by the release of pro-inflammatory cytokines IL-1β and IL-18 1,10 It is a process distinct from apoptosis and necrosis (Table 1) and has been observed in the setting of multiple diseases including, but not limited to, microbial infection 10, atherosclerosis and other
  • Pyroptosis: host cell death and inflammation - PMC
    Pyroptosis, or caspase 1-dependent cell death, is inherently inflammatory, is triggered by various pathological stimuli, such as stroke, heart attack or cancer, and is crucial for controlling microbial infections Pathogens have evolved mechanisms to inhibit pyroptosis, enhancing their ability to persist and cause disease
  • Role of Pyroptosis in inflammatory bowel disease - PubMed
    Pyroptosis, an inflammatory form of programmed cell death activated by inflammasomes, is marked by the rupture of cell membranes and the subsequent release of inflammatory mediators Emerging evidence indicates that pyroptosis plays a crucial role in the pathogenesis of IBD
  • GSDMB is increased in IBD and regulates epithelial . . . - Cell Press
    Our paper showed that stimulated gasdermin B (GSDMB) in its full-length form translocates to the plasma membrane and regulates cell proliferation, migration, and adhesion to promote epithelial restitution and repair independent of pyroptosis





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