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  • Polystyrene microplastics induce gut microbiota dysbiosis and hepatic . . .
    Polystyrene microplastics induce gut microbiota dysbiosis and hepatic lipid metabolism disorder in mice (Deng et al , 2017), there are few studies on the effects of polystyrene MP on the gut microbiota in mice According to previous studies, some The relative abundance of Bacteroidetes decreased after 3 days of 1000 μg L 0 5 and 50 μm
  • Polystyrene microplastics induce size-dependent multi-organ damage in . . .
    For example, a study found that polystyrene (PS) particles with a diameter of 600 nm induced more serious nephrotoxicity than those with diameters of 50 nm and 4 µm [21] In contrast to the tissue damage and the resulting complications caused by small MPs, larger MPs cannot enter the systemic circulation and may instead exert their toxic effects by inducing gut dysbiosis and metabolic disorder
  • Chronic exposure to polystyrene nanoplastics induces intestinal . . .
    Therefore, in this project, we propose a mouse model of polystyrene nanoplastics (PS-NPs) exposure at different concentrations (0 1, 1 and 10 mg·L −1) to (1) clarify the impact of PS-NPs exposure on the intestinal mechanical barrier of mice, (2) identify the intestinal immune cytotoxic effect caused by PS-NPs exposure and (3) assess the health risk of long-term exposure to PS-NPs at
  • Polystyrene microplastics trigger adiposity in mice by remodeling gut . . .
    Polystyrene microplastics induce gut microbiota dysbiosis and hepatic lipid metabolism disorder in mice Sci Total Environ , 631-632 ( 2018 ) , pp 449 - 458 , 10 1016 j scitotenv 2018 03 051 View PDF View article View in Scopus Google Scholar
  • Polystyrene microplastics aggravate inflammatory damage in mice with . . .
    Studies have shown that the accumulated MPs in organisms can promote numerous adverse effects For example, polystyrene microplastics (PSMPs) caused gut epithelial damage and congestive inflammation of zebrafish fluorescence (ex: 615–665 nm, em: 690–770 nm), exposure time (4 s), binning factor (8), f-stop (2), and fields of view (12 5
  • Long-term exposure to polystyrene microplastics reduces macrophages and . . .
    In the microbiota–gut–brain axis, polystyrene MP treatment altered bile acid and carbohydrate metabolism in the intestine, inhibited intestinal motility, reduced water reabsorption, and led to a certain degree of depression in mice Mice were administered three cycles: 6 days of DSS water (2 5 %, w v) followed by 15 days of clean water
  • Oral exposure of polystyrene microplastics and doxycycline affects mice . . .
    Oral intake is a major route for human exposure to MPs; thus, the intestinal tract is one of the principal target organs after MPs’ oral ingestion [2], [9] MPs exposure not only affects the gut microbiota disturbance and metabolic changes but also sabotages the intestinal barrier integrity, which both linked with damage in the distal tissues and organs [10], [11], [12]
  • Polystyrene nanoparticles induced mammalian intestine damage caused by . . .
    The gut microbiota is more sensitive to outer stimuli Thus, the feces of mice were collected after 28-days of exposure to study the effects of PS nanoparticles on the intestine The results showed that even at the low dosage (5 mg·kg −1 ·BW), PS nanoparticles significantly reduced gut microbiota richness of gut microbiota (Fig 7 a, b)
  • Polystyrene micro- and nanoparticles exposure induced anxiety-like . . .
    The mice were exposed to PS-NPs (100 nm, 0 5 mg day) and PS-MPs (1 0 µm, 0 5 mg day) for 60 days Polystyrene microplastics induce gut microbiota dysbiosis and hepatic lipid metabolism disorder in mice Polystyrene microplastics up-regulates liver glutamine and glutamate synthesis and promotes autophagy-dependent ferroptosis and
  • Polystyrene microplastics exacerbated liver injury from . . .
    Polystyrene microplastics exacerbated liver injury from cyclophosphamide in mice: Insight into gut microbiota Author links open we developed a mouse model drinking roughly 18 and 180 μg kg day polystyrene MPs for 90 days, then intraperitoneally injected mice with 80 mg kg cyclophosphamide (CTX) to investigate whether chronic pre-exposure
  • Polystyrene microplastics exposure: Disruption of intestinal barrier . . .
    Moreover, compared with our previous research on intestinal barrier injury induced by a high concentration of PSMPs (500 ppb) in adult mice (Liu et al , 2022a, Liu et al , 2022b), we newly pointed out that PSMPs at a low ambient concentration (20 ppb) still led to impaired gut barrier and liver damage in infant mice, and the results presented the ascending sensitivity of infant mice to MPs





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